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Hai cercato: Magnesium+trisilicate,+hydrate


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Codice catalogo: (BOSSBS-5381R-A750)
Fornitore: Bioss
Codice articolo fornitore: BS-5381R-A750
Codice articolo locale: BOSSBS-5381R-A750
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-3306R-A680)
Fornitore: Bioss
Codice articolo fornitore: BS-3306R-A680
Codice articolo locale: BOSSBS-3306R-A680
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-0222R-A555)
Fornitore: Bioss
Codice articolo fornitore: BS-0222R-A555
Codice articolo locale: BOSSBS-0222R-A555
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-3306R-FITC)
Fornitore: Bioss
Codice articolo fornitore: BS-3306R-FITC
Codice articolo locale: BOSSBS-3306R-FITC
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-0222R-A488)
Fornitore: Bioss
Codice articolo fornitore: BS-0222R-A488
Codice articolo locale: BOSSBS-0222R-A488
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-0222R-A680)
Fornitore: Bioss
Codice articolo fornitore: BS-0222R-A680
Codice articolo locale: BOSSBS-0222R-A680
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-6468R-HRP)
Fornitore: Bioss
Codice articolo fornitore: BS-6468R-HRP
Codice articolo locale: BOSSBS-6468R-HRP
Descrizione: This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-6468R-A488)
Fornitore: Bioss
Codice articolo fornitore: BS-6468R-A488
Codice articolo locale: BOSSBS-6468R-A488
Descrizione: This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-6468R-CY7)
Fornitore: Bioss
Codice articolo fornitore: BS-6468R-CY7
Codice articolo locale: BOSSBS-6468R-CY7
Descrizione: This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-6468R-A350)
Fornitore: Bioss
Codice articolo fornitore: BS-6468R-A350
Codice articolo locale: BOSSBS-6468R-A350
Descrizione: This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-0222R-FITC)
Fornitore: Bioss
Codice articolo fornitore: BS-0222R-FITC
Codice articolo locale: BOSSBS-0222R-FITC
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-3307R-A647)
Fornitore: Bioss
Codice articolo fornitore: BS-3307R-A647
Codice articolo locale: BOSSBS-3307R-A647
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-133 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-0222R-A350)
Fornitore: Bioss
Codice articolo fornitore: BS-0222R-A350
Codice articolo locale: BOSSBS-0222R-A350
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-3306R-A555)
Fornitore: Bioss
Codice articolo fornitore: BS-3306R-A555
Codice articolo locale: BOSSBS-3306R-A555
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-5380R-HRP)
Fornitore: Bioss
Codice articolo fornitore: BS-5380R-HRP
Codice articolo locale: BOSSBS-5380R-HRP
Descrizione: NMDA receptor subtype of glutamate-gated ion channels with high calcium permeability and voltage-dependent sensitivity to magnesium. Mediated by glycine. In concert with DAPK1 at extrasynaptic sites, acts as a central mediator for stroke damage. Its phosphorylation at Ser-1303 by DAPK1 enhances synaptic NMDA receptor channel activity inducing injurious Ca2+ influx through them, resulting in an irreversible neuronal death (By similarity).
UOM: 1 * 100 µl


Codice catalogo: (BOSSBS-6468R-A680)
Fornitore: Bioss
Codice articolo fornitore: BS-6468R-A680
Codice articolo locale: BOSSBS-6468R-A680
Descrizione: This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterised by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium (By similarity).
UOM: 1 * 100 µl


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La disponibilità per questo articolo è limitata, ma potrebbe essere disponibile in un magazzino vicino a voi. Si prega di assicurarsi che si è effettuato l'accesso al sito, in modo che ledisponibilità possano essere visualizzati. Se il call è ancora visualizzato e hai bisogno di assistenza, si prega di telefonare a 1-800-932 - 5000.
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